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K., Mandel M., Barron E

We established an FP competition assay to biochemically validate displacement of the TIN2 peptide by the compounds and further showed that compound 40 , identified as one of the most potent analogues amongst the compounds tested, evicted full-length TRF1 from the shelterin complex by disrupting the TRF1 TRFH :TIN2 TBM interaction

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2C), but the quantity of autophagosomes was lessen in TAC + semaglutide + HCQ group by TEM, compared to those in TAC + SMGLT group (Fig

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