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[DOI] [PubMed] [Google Scholar] 119.Madayag A., Lobner D., Kau K.S., Mantsch J.R., Abdulhameed O., Hearing M., Grier M.D., Baker D.A

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If there is a deficiency of G6PH, there will not be enough NADPH to reduce GSSG, leading to decreased levels of GSH

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Experimental and translational studies suggest that elevated prolactin levels contribute to obesity-related phenotypes through multiple pathways, including altered central appetite regulation, modulation of adipocyte differentiation and lipid storage, impairment of insulin sensitivity, and disruption of the hypothalamicpituitarygonadal (HPG) axis