slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis slc25a39 is necessary for mitochondrial
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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39 mediated transport Dual regulation of SLC25A39 by Controlling glutathione entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease Signal Transduction and Targeted Therapy SLC25A39 links mitochondrial GSH sensing with iron metabolism: Molecular Cell Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis: Molecular Cell SLC25A39 identified as a key regulator of hepatocellular carcinoma progression through the mitochondrial ROScytochrome ccaspase signaling axis Cellular & Molecular Biology Letters Springer Nature Link
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