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The sexual function effects of MT-2 are centrally mediated through hypothalamic melanocortin circuits, not through peripheral vascular mechanisms, which distinguishes MT-2 from PDE5 inhibitors and explains its effectiveness in patients where desire and arousal motivation, rather than vascular mechanics, is the limiting factor

We summarize key mechanisms of action, categorize clinically relevant peptide classes, and address current limitations, including gaps in human data, safety considerations, and regulatory ambiguity

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Tryptophan depletion increases nausea, headache and photophobia in migraine sufferers